Vitamin D Deficiency May Trigger Multiple Sclerosis, Study Finds – iTech Post
The Lack Of Vitamin D Levels Is Found To Contribute To Increase The Risks Of Developing Multiple Sclerosis, But How Is That Possible? Find Out What Experts Have To Say
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A recent study looking at pregnant women in Finland has been reportedly showing a probable between low levels of vitamin D and the development of multiple sclerosis. It was found that there is an increasing when it comes to the number of evidence that vitamin D may have a crucial part in Multiple Sclerosis.
Multiple Sclerosis And The Role Of Vitamin D
As of the present time, the said kind of illness has already affected an estimated 2.3 millions of individuals all over the world which has been diagnosed with Relapsing-Remitting Multiple Sclerosis (RRMS). On the other hand, a new research has also suggested the introduction of “alemtuzumab” as a means to improve some of the physical inabilities linked to the disease.
MD Magazine reveals that the study has been conducted through a repository with over 1.5 million blood samples collected in Finland from all the pregnant women since 1983. The level of vitamin D collected from healthy women can be related to their future risk of developing MS.
Furthermore, Multiple Sclerosis Today has revealed that a promising research from the Cambridge Center for Myelin Repair has found that vitamin D may play a role in myelin repair.The study shows that vitamin D can be involved in the regeneration of myelin from the onset of the disease.
Experts have highly emphasized that early detection of the said illness may include weakness, tingling, numbness, and blurred vision. Other signs are muscle stiffness, inability to think well , and urinary problems. However, for the study, the researchers conclude that those women who were vitamin deficient at the time of pregnancy had a twofold higher risk of developing MS.
The cause of multiple sclerosis still remains to be unknown, however, scientists believe the disease is triggered by as-yet-unidentified environmental factor in a person who is genetically predisposed to respond.